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OILY SKIN AND HYPERSEBORRHEA

Download a sample report

Oily skin is characterized by the accumulation on its surface of excess sebum, a substance secreted by sebaceous glands. Sebum is composed of triglycerides, diglycerides and monoglycerides (fatty acids linked to a glycerol motif), wax esters, squalene (very sensitive to oxidation) and sterols (eg. cholesterol). Sebum is secreted by sebocytes through a process of cell differentiation and lipid synthesis or accumulation, called lipidogenesis.

Lipidogenesis is dependent on many factors (hormones, food, pharmaceutical active ingredients, xenobiotics, etc.), which can be identified in in vitro experiments. Mature sebocytes undergo apoptosis and burst to release sebum, which travels through the hair follicle to the skin surface. Sebum lubricates the hair follicle and spreads over the surface of the skin at the stratum corneum level.

An overproduction of sebum, called hyperseborrhea, is responsible for oily skin and greasy hair. In some cases, hyperseborrhea can also contribute to the development of acne prone skin.

SEBO662-3D (lipids in red - Oil-Red-O)
SEBO662 (lipids in green-bodipy)
SEBO662 (lipids in green, cytokeratin 7 in red)
SEBO662 (lipids in green, cytokeratin 7 in red)

Oily skin and hyperseborrhea: models and assays

in silico assays

in vitro et ex vivo assays
& oily skin

Biochemical analysis
& oily skin

Clinical imaging
& oily skin

in vitro models & assays

QIMA Life Sciences has many in vitro or ex vivo models at your disposal:

  • sebocyte cell line (SEBO662)
  • 3D sebocyte cell line (SEBO662 3D)
  • sebocyte cell line in response to androgens (SEBO662AR)

on which we can evaluate the effects of active ingredients or cosmetic formulations on sebaceous gland regulation and acne by measuring:

  • sebocyte differentiation and maturation  (EMA, KRT7, etc.)
  • lipidogenesis (lipid accumulation and production)
  • 5-alpha reductase activity and testosterone metabolism

Here are a few examples among all assays proposed by QIMA Life Sciences in the field of oily skin and hyperseborrhea:

No products found which match your selection.

Biochemical analysis of non-invasive clinical samples

Analysis of sebum components and free fatty acids

Our company has developed ready-to-use non-invasive collection kits to analyze the surface lipids and biomarkers of the skin from your samples, or from those of your clinical center.

The sebaceous lipids and the fatty acids that constitute the epidermis and scalp are sampled using two non-invasive sampling kits (SB Kit and SW Kit).

The analysis of the components of sebum and free fatty acids makes it possible to assess:

  • The lipase activity of certain microorganisms in the skin microbiota
  • Sebum regulation
  • Inflammation – by quantification of pro / anti-inflammatory fatty acids
  • Oxidative stress – peroxidation (peroxidized squalene), detoxification (CAT, SOD)
  • Anti-microbial activity
  • The qualitative and quantitative composition of sebum

These analyses help support your claims about the efficacy of anti-acne, oily skin, soothing, protective, anti-pollution products, etc.

Sebaceous lipids screening

Detailed composition of sebum fatty acids by CG/SM

Skin microbiota and inflammation analysis

Hyperseborrhea is frequently associated with disorders of the skin microbiota. Maintaining a good balance in the microbiota helps minimize the inflammation caused by the bacteria that colonize the hair follicle (e.g. C. acnes).

Samples are taken using the SW Kit. The qualitative and quantitative analyses of the bacteria present on the surface of the skin are carried out by:

  • Traditional microbiology on agar
  • Targeted qPCR
  • Non-targeted metagenomic analysis

Inflammatory markers (cytokines, fatty acids, PGE2) are also analyzed.

These analyses help support your claims about the efficacy of “friendly” microbiota, bacteriostatic, prebiotic, soothing products, etc.

Data mapping and clinical imaging

Oily and acne-prone skin presents many markers that can be monitored by 2D color imaging.

Measurement of skin imperfections and of oily skin inflammation in 2D color imaging

Acne lesions can be detected by image analysis, by using powerful algorithms that can discriminate inflammatory lesions from non-inflammatory lesions, or post-inflammatory pigmentation.
The monitoring of inflammation parameters allows the effectiveness of acne skincare products to be measured, either on the whole face or locally by macro imaging, and for short-range or long-range kinetics.
The density of lesions, their total surface and their color are all markers of their evolution.

Acquisition d’image du visage entier sans contrainte avec le ColorFace®

ColorFace®

Acquisition and analysis of acneic lesions and of inflammation on images acquired with ColorFace®

Analyse de PIH en visage entier sur images acquises au ColorFace ®

Analysis of PIH of whole face on images acquired with ColorFace®

Newtone SkinCam noire 010

SkinCam

Analysis and monitoring of lesions on images acquired with SkinCam® 

Measurement of acne lesions and inflammation by hyperspectral imaging

Newtone’s hyperspectral imaging is a unique technology developed to calculate the skin chromophore absorption mappings, and consequently to measure evolution over time, while providing a perfectly representative image of the measured evolution.
Acquisition systems allow image acquisition of the whole face (SpectraFace) or of a defined area of the body and face (SpectraCam).
On these images, hemoglobin can be monitored and analyzed as by color imaging, while discriminating it from melanin. On these images, the same parameters as in 2D color imaging can be extracted for each skin lesion, in addition to hemoglobin concentration.

Spectracam

Spectracam

Acquisition and analysis of acneic lesions and inflammation on images acquired with SpectraCam®

Measurement of oily skin gloss linked to seborrhea in 2D color imaging

Oily skin gloss is measured by calculating the quantity of specular reflection. A gloss map extraction and the monitoring of oily skin glossiness allow sebum evolution to be monitored and anti-seborrheic and mattifying effects to be evaluated.
The monitoring of glossiness parameters allows the effectiveness of oily skin care products to be measured, either on the whole face or locally by macro imaging, and for short-range or long-range kinetics.

Acquisition d’image du visage entier sans contrainte avec le ColorFace®

ColorFace®

Measurement of gloss map of cheek on image acquired with ColorFace®

Measurement of skin fluorescence on image acquired with ColorFace ®

Measurement of pore visibility on image acquired with ColorFace®

Measurement of porphyrin fluorescence in UV imaging

Acne-prone skin can present a high fluorescence rate under UV, due to the metabolism of the bacteria specific to acne. The measurement of porphyrin fluorescence intensity is therefore a marker of the evolution of the inflammatory and bacterial skin condition.

Measurement of pore visibility in 2D imaging

One of the characteristics of oily skin is an increased pore visibility, which is linked to pore dilation and obstruction, as well as to the glossiness induced by seborrhea.
Pore analysis can be carried out on images in parallel and cross polarized light, according to the required method.
Parameters for surface, for detected pore density and for visible depth are all markers to be monitored in order to investigate the astringent effect of the treatment.

Find out about all our standard assays - Direct access to catalog
A project, an idea or a question? Contact us and let’s talk about it!

Oily skin and hyperseborrhea: posts and publications

Innate immunity activation of sebocyte cells by living bacteria. Evidence of a potential immunosuppressive effect of DHT.

Acne, Microbiome cutané, Sebaceous gland regulation, Skin microbiome, Skin microbiome

Acne is a skin pathology targeting the pilosebaceous unit…sebum hypersecretion and bacterial infection. We studied the effects of living bacteria on the induction of an innate immune response in a sebocyte cell line.

Read more
https://qima-lifesciences.com/wp-content/uploads/2019/05/190527_Poster1_petit.png 368 655 Vanessa https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Vanessa2019-05-22 14:37:542019-11-12 17:58:17Innate immunity activation of sebocyte cells by living bacteria. Evidence of a potential immunosuppressive effect of DHT.

Pharmacological effects of anti-androgens and other compounds in an androgen-sensitive sebocyte cell line.

Acne, Sebaceous gland regulation

We analysed the activity of reference anti-androgens (Finasteride, Dutasteride, Cyproterone acetate), at the level of their potential targets (5-alpha-reductase, AR translocation, induced transcripts) and in a functional lipid accumulation assay. We also evaluated this androgen-induced lipid accumulation assay as a potential pharmacological tool to detect potential inhibitors from both androgenic and non-androgenic origins.

Read more
https://qima-lifesciences.com/wp-content/uploads/2019/05/190527_Poster3_petit.png 368 655 Vanessa https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Vanessa2019-05-22 14:37:122019-11-12 17:36:48Pharmacological effects of anti-androgens and other compounds in an androgen-sensitive sebocyte cell line.

Androgens activate lipogenesis through an AKT-independent mTOR pathway stimulation and a limitation of autophagy in an androgen-sensitive sebocyte cell line.

Acne, Peau grasse, hyperséborrhée et séborégulation, Sebaceous gland regulation

Androgens are key regulators of sebaceous function… A link between mTOR and androgen signaling has previously been reported…as well as a link between these processes and lipid synthesis…Here we aimed at confirming that DHT-induced lipid synthesis/accumulation is at least in part dependent on mTOR activation in the SEBO662AR cell line and that the autophagic process is consequently modified by the androgenic treatment.

Read more
https://qima-lifesciences.com/wp-content/uploads/2019/05/190527_Poster4_petit.png 368 655 Vanessa https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Vanessa2019-05-22 14:37:042019-11-12 17:39:16Androgens activate lipogenesis through an AKT-independent mTOR pathway stimulation and a limitation of autophagy in an androgen-sensitive sebocyte cell line.
lipid

Effects of different kinds of pollutants on the lipidic metabolism of human sebocytes and protective effects of a new macroalgae culture extract

Cosmetics, Peau grasse, hyperséborrhée et séborégulation, Sebaceous gland regulation, Skin protection and immune defense system

Sebocytes lipid production are stimulated by pollutants. Ame acts by protecting epidermal cells, human sebocytes and skin from urban dust.

Read more
https://qima-lifesciences.com/wp-content/uploads/2017/11/SEPPIC-Image-à-la-une.jpg 640 1140 Guillaume https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Guillaume2017-11-29 10:57:192019-09-13 07:39:35Effects of different kinds of pollutants on the lipidic metabolism of human sebocytes and protective effects of a new macroalgae culture extract

Acne vulgaris: physiopathology and cell mechanisms

Acne, Sebaceous gland regulation

Acne Vulgaris is a chronic inflammatory pathology located in the region of the pilosebaceous follicle. It occurs following a high production of sebum linked to hormonal (androgynous) or environmental (pollution) malfunctioning and to the colonization of the pilosebaceous follicle by certain bacteria (P. acnes).
QIMA Life Sciences has developed a panel of innovative assays to discover and evaluate compounds for the treatment of acne.

Read more
https://qima-lifesciences.com/wp-content/uploads/2017/11/Image-à-la-une.jpg 368 655 Julie https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Julie2017-11-29 09:30:252019-09-13 07:40:20Acne vulgaris: physiopathology and cell mechanisms

Physiology and functions of the sebaceous gland

Acne, Sebaceous gland regulation, Skin barrier and hydration

The sebaceous gland is an organ located in the dermis. Its role is to synthesise and secrete sebum which is a component of the hydrolipidic film. The purpose of sebum is to protect the skin from external aggression and dehydration. Sebum also maintains the suppleness of the skin and hair.

Read more
https://qima-lifesciences.com/wp-content/uploads/2017/11/Sébocytes-image-à-la-une.jpg 368 701 Julie https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Julie2017-11-28 09:24:342019-11-07 15:45:07Physiology and functions of the sebaceous gland

Androgens induce sebaceous differentiation in sebocyte cells expressing a stable functional androgen receptor

Acne, Cell and tissue engineering, Sebaceous gland regulation

In order to study the impact of active androgens in sebocytes, we constructed a stable human sebocyte cell line derived from SEBO662 [17] constitutively expressing a fully functional AR. In these SEBO662 AR+ cells, dihydrotestosterone (DHT) induced AR nuclear translocation and the strong modulation of a set of transcripts (RASD1, GREB1…) known to be androgen-sensitive in other androgenic cells and tissues.

Read more
https://qima-lifesciences.com/wp-content/uploads/2016/03/mw_androgens-induce-sebaceous-differentiation-in-sebocyte.jpg 368 655 Guillaume https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Guillaume2015-04-09 15:47:492019-11-13 11:16:58Androgens induce sebaceous differentiation in sebocyte cells expressing a stable functional androgen receptor

Immortalized sebocytes SEBO662 can spontaneously differentiate into a sebaceous-like phenotype when cultured as a 3D epithelium

Acne, Cell and tissue engineering, Sebaceous gland regulation

SEBO662 multilayers spontaneously differentiate into a sebaceous-like structure as shown by the strong polarized expression of the late sebaceous marker EMA, the overexpression of some lipogenic markers and lipid production on the upper side of the epithelium.

Read more
https://qima-lifesciences.com/wp-content/uploads/2016/05/mw_Immortalized-sebocytes.jpg 368 655 Guillaume https://qima-lifesciences.com/wp-content/uploads/2021/10/QIMA_life_sciences@4x1.png Guillaume2012-01-24 10:17:152019-11-13 11:28:55Immortalized sebocytes SEBO662 can spontaneously differentiate into a sebaceous-like phenotype when cultured as a 3D epithelium

OILY SKIN AND HYPERSEBORRHEA

Download a sample report

Address

QIMA Life Sciences
Headquarters
1bis rue des plantes - CS 50011
86160 Gençay - France
Tel. +33 (0) 5 49 36 11 37
Mail: ls@qima.com

International offices
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Latest posts

  • Performance and Tolerance of a Protocol for Idiopathic Chronic Greasy Seborrhea in 18 Dogs Using a Shampoo and Mousse Containing Plant Extracts
  • Evaluation of a new connected portable camera for the analysis of skin microrelief and the assessment of the effect of skin moisturisers
  • Clinical and Microbiological Performances and Effects on Lipid and Cytokine Production of a Ceruminolytic Ear Cleaner in Canine Erythemato-Ceruminous Otitis Externa

Cosmetic

  • Hydration and skin barrier
  • Epidermal regeneration
  • Skin firmness & cohesion
  • Skin ageing
  • Skin protection & defense
  • Skin pigmentation
  • Oily skin and hyperseborrhea
  • Skin microcirculation and vascularization
  • Slimming and adipocyte metabolism
  • Hair growth and alopecia

Pharmacology

  • Immuno-Inflammation
  • Neurobiology
  • Veterinary medicine

Dermatology

  • Acne
  • Hair growth and alopecia
  • Atopic dermatitis
  • Psoriasis
  • Wound healing and skin regeneration
  • Melanoma
  • Vitiligo (coming soon)
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Ressources documentaires


  • Les informations recueillies sur ce formulaire sont enregistrées dans un fichier informatisé par QIMA LIFE SCIENCES pour vous contacter afin de répondre à votre demande et vous aider à sélectionner le ou les tests les mieux adaptés à votre projet.

  • La base légale du traitement est l’intérêt légitime du QIMA Life Sciences. Les données collectées seront communiquées aux seuls destinataires suivants : Les services concernés et habilités par QIMA Life Sciences. Les données sont conservées pendant une durée de 3 ans à compter de la collecte ou de votre dernier contact entrant.

    Vous pouvez accéder aux données vous concernant, les rectifier, demander leur effacement ou exercer votre droit à la limitation du traitement de vos données, retirer votre consentement au traitement de vos données à tout moment, vous opposer au traitement de vos données et vous disposez d’un droit à la portabilité de vos données.

    Consultez le site cnil.fr pour plus d’informations sur vos droits.

    Pour exercer ces droits vous pouvez nous contacter à l’aide du lien suivant Gestion des droits ou en écrivant à dpo-qima@agencergpd.eu.

    Si vous estimez, après nous avoir contactés, que vos droits « Informatique et Libertés » ne sont pas respectés, vous pouvez adresser une réclamation à la CNIL.

    N.B : Les catégories de données suivantes : Email / Pays sont obligatoires. La non-fourniture de ces informations entrainera l’impossibilité pour QIMA Life Sciences de vous répondre.


  • This field is for validation purposes and should be left unchanged.

Documentary Ressources_Flyer_Ceramides


  • Les informations recueillies sur ce formulaire sont enregistrées dans un fichier informatisé par QIMA LIFE SCIENCES pour vous contacter afin de répondre à votre demande et vous aider à sélectionner le ou les tests les mieux adaptés à votre projet.

  • La base légale du traitement est l’intérêt légitime du QIMA Life Sciences. Les données collectées seront communiquées aux seuls destinataires suivants : Les services concernés et habilités par QIMA Life Sciences. Les données sont conservées pendant une durée de 3 ans à compter de la collecte ou de votre dernier contact entrant.

    Vous pouvez accéder aux données vous concernant, les rectifier, demander leur effacement ou exercer votre droit à la limitation du traitement de vos données, retirer votre consentement au traitement de vos données à tout moment, vous opposer au traitement de vos données et vous disposez d’un droit à la portabilité de vos données.

    Consultez le site cnil.fr pour plus d’informations sur vos droits.

    Pour exercer ces droits vous pouvez nous contacter à l’aide du lien suivant Gestion des droits ou en écrivant à dpo-qima@agencergpd.eu.

    Si vous estimez, après nous avoir contactés, que vos droits « Informatique et Libertés » ne sont pas respectés, vous pouvez adresser une réclamation à la CNIL.

    N.B : Les catégories de données suivantes : Email / Pays / Société / Sélectionnez la vidéo sont obligatoires. La non-fourniture de ces informations entrainera l’impossibilité pour QIMA Life Sciences de vous répondre.


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